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Osteonecrosis of the jaw (ONJ) is a severe bone disease (
osteonecrosis) that affects the
jaws (the maxilla and the mandible).Various forms of ONJ have been described over the last 160 years, and a number of causes have been suggested in the literature.
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Zoledronic acid) (Zometa, Reclast), a member of the
bisphosphonate class, inhibits osteoclast-mediated bone resorption. The antiresorptive mechanism is not fully understood and several factors are thought to contribute to this action.
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Osteonecrosis of the jaw (ONJ), which was first described in 2002 [], is a relatively uncommon but potentially serious side effect of
treatment with antiresorptive agents such as
intravenous (IV) high potency bisphosphonates and denosumab, which decrease the risk of skeletal-related events in patients with cancer and metastatic bone disease.
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osteonecrosis of the jaw (MON, MRONJ) is progressive death of the jawbone in a person exposed to a medications known to increase the risk of disease, in the absence of a previous radiation
treatment. It may lead to surgical complication in the form of impaired wound healing following
oral and maxillofacial surgery, periodontal surgery, or endodontic therapy.
Adverse drug events in the oral cavity are common and will likely increase as newer therapeutic agents are approved. Health care providers should familiarize themselves with such events.
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Avascular necrosis (AVN) is defined as cellular death of bone components due to interruption of the blood supply; the bone structures then collapse, resulting in pain, loss of joint function and long-term joint damage. Ebook Oral And Intravenous Bisphosphonate Induced Osteonecrosis Of The Jaws History Etiology Prevention And Treatment Kindle Autologous Platelet Concentrate. Lopez-Jornet and colleagues (2016) performed a systematic literature review to determine the effectiveness of autologous platelet concentrate (APC) application, for
prevention or
treatment of medication-related
osteonecrosis of the …
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